Rheumatoid arthritis (RA) may be an autoimmune disease, but its collateral damage โ erosion of cartilage, joint deformity, and synovial hypertrophy โ lands squarely in orthopedic territory.
And now, a new small-molecule contender is entering the race to protect those joints before surgeons ever need to replace them.
Subscribe to continue reading
- Unlimited access to our content and archive
- Exclusive access to our newsletter
- Join the Conversation! Exclusive access to article comments.

