Rotator cuff tears are a familiar frustration for shoulder surgeons. Repair the tendon perfectly, and the muscle still has the final word. Atrophy, fatty infiltration, and fibrosis can quietly undermine even technically flawless surgery, leaving the repaired cuff weaker than hoped.
A new laboratory study suggests an unexpected ally might help: blood flow restriction (BFR) therapy — and the tiny cellular power plants known as mitochondria.
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